Conference Paper
The Non‐Uniqueness of the ITS‐90: 13.8033 K to 273.16 K
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AIP Conf. Proc. 684, 53 (Mon Sep 29 00:00:00 UTC 2003);
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21-24 October 2002
Chicago, Illinois (USA)
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Estimates of the non‐uniqueness of the ITS‐90 are reported based on comparisons of capsule‐style standard platinum resistance thermometers at more than eighty temperatures between 13.8033 K and 273.16 K. Using the measurements reported here, as well as those of Ward and Compton and Head, we conclude that the non‐uniqueness takes on its largest value (± 0.4 mK) in the range from 83.8058 K to 234.3156 K. This result may have been anticipated due to the fact that the argon and mercury fixed points are separated in temperature by more than 150 K, with no intermediate calibration points. Discussions concerning the details of the temperature scale that will eventually replace the ITS‐90 must consider this fact if the non‐uniqueness is to be minimized. At the present time, however, there are no candidate fixed points within this temperature range that are realizable to the required level of accuracy for inclusion into a revised International Temperature Scale.
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The Non‐Uniqueness of the ITS‐90: 13.8033 K to 273.16 K
AIP Conf. Proc. 684, 53 (Mon Sep 29 00:00:00 UTC 2003);
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Scitation: The Non‐Uniqueness of the ITS‐90: 13.8033 K to 273.16 K
http://aip./content/aip/proceeding/aipcp/10.7100
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proceedings.aip.org/684/10.7100/Right1,Right2,Right3Original Article
Korean Journal of Preventive Medicine ): 11-24.
HgCl2 Dysregulates the Immune Response of Balb/c Mice.
1Department of Preventive Medicine, College of Medicine, Chonbuk University, Korea. 2Department of Microbiology, College of Medicine, Chonbuk University, Korea.
The studies reported here were undertaken to investigate the effects of mercury chloride on immune system of Balb/c mouse employing a flexible tier of in vitro and in vivo assays. Mercury chloride inhibited the proliferative responses of spleen cells to lipopolysaccharide, pokeweed mitogen, and phytohemagglutinin as a dose-dependent manner. This inhibitory effect was observed not only when HgCl2 was added 2nd or 3rd day of 3 days culture period but also when spleen cells was pretreated with HgC12 for 2 hours. Mercury chloride, however, potentiated the production of IgM and IgG from spleen cells. During the HgCl2 administration by drinking for 3 weeks, the weight gain of mice was significantly blunted than that of control group mice, while no overt signs related to mercury toxicity were noted in any mice of experimental group. There was no change in thymus and spleen weights, and in histological findings of kidney, bone marrow of femur, thymus, spleen, and politeful lymph node after 3 weeks of mercury exposure. However, HgC12 induced a significant increase of total serum IgM, IgG including IgG1, IgG2a and IgG2b, and IgE in Balb/c mice. Treatment in vivo with anti-IL-4 monoclonal antibody significantly abrogated the HgCl2-induced increase in total serum IgG1 and IgE. Whereas HgCl2 potentiated total serum IgM and IgG, there was, there was no difference in total serum hemagglutinin to SRBC(Sheep Red Blood cell) between experimental and control group mice when these mice were immunized with SRBC. All these findings observed in Balb/c mice suggest that mercury perturbates well-orchestrated regulation of immune responses before developing histopathological changes in lymphoid tissues.
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